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rabbit polyclonal anti cd34 antibody  (Affinity Biosciences)

 
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    Affinity Biosciences rabbit polyclonal anti cd34 antibody
    Exosomal miR-144-3p involved in PPT regulation and MCD in response to neurogenic inflammation and peripheral nociceptor hypersensitivity. ( A ) Immunohistochemical staining images showing colocalization of <t>CD34</t> (MCs; red) and PGP9.5 (axons; green). Cell nuclei were stained with DAPI (blue). ( B ) Quantitative analyses of the colocalization of CD34 (MCs) and PGP9.5 (axons) (n = 3 per group). ( C ) The ratio of the number of cells expressing both CD34 (MCs) and PGP9.5 (axons) to the number of CD34 + MCs (n = 3 per group). All data are shown as the mean ± SD, ∗∗ P < 0.01, ## P < 0.01.
    Rabbit Polyclonal Anti Cd34 Antibody, supplied by Affinity Biosciences, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+polyclonal+anti-cd34+antibody/anti+cd34/pmc12902286-96-9-13
    Average 86 stars, based on 1 article reviews
    rabbit polyclonal anti cd34 antibody - by Bioz Stars, 2026-10
    86/100 stars

    Images

    1) Product Images from "Plasma-derived exosomal miR-144-3p targeting IL-1β is involved in pressure pain threshold regulation at PC6 acupoint in myocardial ischemia rats"

    Article Title: Plasma-derived exosomal miR-144-3p targeting IL-1β is involved in pressure pain threshold regulation at PC6 acupoint in myocardial ischemia rats

    Journal: Journal of Traditional and Complementary Medicine

    doi: 10.1016/j.jtcme.2025.02.001

    Exosomal miR-144-3p involved in PPT regulation and MCD in response to neurogenic inflammation and peripheral nociceptor hypersensitivity. ( A ) Immunohistochemical staining images showing colocalization of CD34 (MCs; red) and PGP9.5 (axons; green). Cell nuclei were stained with DAPI (blue). ( B ) Quantitative analyses of the colocalization of CD34 (MCs) and PGP9.5 (axons) (n = 3 per group). ( C ) The ratio of the number of cells expressing both CD34 (MCs) and PGP9.5 (axons) to the number of CD34 + MCs (n = 3 per group). All data are shown as the mean ± SD, ∗∗ P < 0.01, ## P < 0.01.
    Figure Legend Snippet: Exosomal miR-144-3p involved in PPT regulation and MCD in response to neurogenic inflammation and peripheral nociceptor hypersensitivity. ( A ) Immunohistochemical staining images showing colocalization of CD34 (MCs; red) and PGP9.5 (axons; green). Cell nuclei were stained with DAPI (blue). ( B ) Quantitative analyses of the colocalization of CD34 (MCs) and PGP9.5 (axons) (n = 3 per group). ( C ) The ratio of the number of cells expressing both CD34 (MCs) and PGP9.5 (axons) to the number of CD34 + MCs (n = 3 per group). All data are shown as the mean ± SD, ∗∗ P < 0.01, ## P < 0.01.

    Techniques Used: Immunohistochemical staining, Staining, Expressing

    Related Articles

    other:

    Article Title: Plasma-Derived Exosomal miR-144-3p Targeting IL-1β is Involved in Pressure Pain Threshold Regulation at PC6 Acupoint in Myocardial Ischemia Rats
    Article Snippet: The sections were 230 incubated with primary antibodies including a rabbit polyclonal anti-CD34 antibody (Affinity 231 Biosciences, Cincinnati, Ohio, USA) and rabbit polyclonal anti-Protein gene product 9.5 (PGP9.5) 232 antibody (Affinity Biosciences, Cincinnati, Ohio, USA).



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    Exosomal miR-144-3p involved in PPT regulation and MCD in response to neurogenic inflammation and peripheral nociceptor hypersensitivity. ( A ) Immunohistochemical staining images showing colocalization of <t>CD34</t> (MCs; red) and PGP9.5 (axons; green). Cell nuclei were stained with DAPI (blue). ( B ) Quantitative analyses of the colocalization of CD34 (MCs) and PGP9.5 (axons) (n = 3 per group). ( C ) The ratio of the number of cells expressing both CD34 (MCs) and PGP9.5 (axons) to the number of CD34 + MCs (n = 3 per group). All data are shown as the mean ± SD, ∗∗ P < 0.01, ## P < 0.01.
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    Immunofluorescence and immunohistochemical analysis in fistula tissues. (A) Representative immunofluorescence staining of <t>CD34</t> (red) and DAPI (blue) in fistula tissues from Control, PRP, and Cryo-PRP groups. (B) Quantitative analysis of CD34 expression using integrated optical density (IOD/Area). (C) Representative images of immunohistochemical staining for α-SMA, CD31, VEGF, PCNA, and TNF-α in the Control, PRP, and Cryo-PRP groups are shown (original magnification: ×400). Expression of α-SMA, CD31, VEGF, and PCNA was markedly increased in the PRP and Cryo-PRP groups compared to Control, with the highest levels observed in the Cryo-PRP group. In contrast, TNF-α expression was significantly reduced following PRP and Cryo-PRP treatment, with minimal staining in the Cryo-PRP group. (D) Quantitative analysis of immunohistochemical staining using integrated optical density per area (IOD/Area × 10 6 ). Data are presented as mean ± SD. * P < 0.05 vs. Control; *** P < 0.001 vs. Control.
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    Immunofluorescence and immunohistochemical analysis in fistula tissues. (A) Representative immunofluorescence staining of <t>CD34</t> (red) and DAPI (blue) in fistula tissues from Control, PRP, and Cryo-PRP groups. (B) Quantitative analysis of CD34 expression using integrated optical density (IOD/Area). (C) Representative images of immunohistochemical staining for α-SMA, CD31, VEGF, PCNA, and TNF-α in the Control, PRP, and Cryo-PRP groups are shown (original magnification: ×400). Expression of α-SMA, CD31, VEGF, and PCNA was markedly increased in the PRP and Cryo-PRP groups compared to Control, with the highest levels observed in the Cryo-PRP group. In contrast, TNF-α expression was significantly reduced following PRP and Cryo-PRP treatment, with minimal staining in the Cryo-PRP group. (D) Quantitative analysis of immunohistochemical staining using integrated optical density per area (IOD/Area × 10 6 ). Data are presented as mean ± SD. * P < 0.05 vs. Control; *** P < 0.001 vs. Control.
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    Image Search Results


    Exosomal miR-144-3p involved in PPT regulation and MCD in response to neurogenic inflammation and peripheral nociceptor hypersensitivity. ( A ) Immunohistochemical staining images showing colocalization of CD34 (MCs; red) and PGP9.5 (axons; green). Cell nuclei were stained with DAPI (blue). ( B ) Quantitative analyses of the colocalization of CD34 (MCs) and PGP9.5 (axons) (n = 3 per group). ( C ) The ratio of the number of cells expressing both CD34 (MCs) and PGP9.5 (axons) to the number of CD34 + MCs (n = 3 per group). All data are shown as the mean ± SD, ∗∗ P < 0.01, ## P < 0.01.

    Journal: Journal of Traditional and Complementary Medicine

    Article Title: Plasma-derived exosomal miR-144-3p targeting IL-1β is involved in pressure pain threshold regulation at PC6 acupoint in myocardial ischemia rats

    doi: 10.1016/j.jtcme.2025.02.001

    Figure Lengend Snippet: Exosomal miR-144-3p involved in PPT regulation and MCD in response to neurogenic inflammation and peripheral nociceptor hypersensitivity. ( A ) Immunohistochemical staining images showing colocalization of CD34 (MCs; red) and PGP9.5 (axons; green). Cell nuclei were stained with DAPI (blue). ( B ) Quantitative analyses of the colocalization of CD34 (MCs) and PGP9.5 (axons) (n = 3 per group). ( C ) The ratio of the number of cells expressing both CD34 (MCs) and PGP9.5 (axons) to the number of CD34 + MCs (n = 3 per group). All data are shown as the mean ± SD, ∗∗ P < 0.01, ## P < 0.01.

    Article Snippet: The sections were incubated with primary antibodies including a rabbit polyclonal anti-CD34 antibody (Affinity Biosciences, Cincinnati, Ohio, USA) and rabbit polyclonal anti-Protein gene product 9.5 (PGP9.5) antibody (Affinity Biosciences, Cincinnati, Ohio, USA).

    Techniques: Immunohistochemical staining, Staining, Expressing

    Immunofluorescence and immunohistochemical analysis in fistula tissues. (A) Representative immunofluorescence staining of CD34 (red) and DAPI (blue) in fistula tissues from Control, PRP, and Cryo-PRP groups. (B) Quantitative analysis of CD34 expression using integrated optical density (IOD/Area). (C) Representative images of immunohistochemical staining for α-SMA, CD31, VEGF, PCNA, and TNF-α in the Control, PRP, and Cryo-PRP groups are shown (original magnification: ×400). Expression of α-SMA, CD31, VEGF, and PCNA was markedly increased in the PRP and Cryo-PRP groups compared to Control, with the highest levels observed in the Cryo-PRP group. In contrast, TNF-α expression was significantly reduced following PRP and Cryo-PRP treatment, with minimal staining in the Cryo-PRP group. (D) Quantitative analysis of immunohistochemical staining using integrated optical density per area (IOD/Area × 10 6 ). Data are presented as mean ± SD. * P < 0.05 vs. Control; *** P < 0.001 vs. Control.

    Journal: Frontiers in Bioengineering and Biotechnology

    Article Title: A novel cryoprecipitate-enriched PRP (Cryo-PRP) gel with enhanced mechanical strength and regenerative capacity accelerates enterocutaneous fistula healing

    doi: 10.3389/fbioe.2025.1668608

    Figure Lengend Snippet: Immunofluorescence and immunohistochemical analysis in fistula tissues. (A) Representative immunofluorescence staining of CD34 (red) and DAPI (blue) in fistula tissues from Control, PRP, and Cryo-PRP groups. (B) Quantitative analysis of CD34 expression using integrated optical density (IOD/Area). (C) Representative images of immunohistochemical staining for α-SMA, CD31, VEGF, PCNA, and TNF-α in the Control, PRP, and Cryo-PRP groups are shown (original magnification: ×400). Expression of α-SMA, CD31, VEGF, and PCNA was markedly increased in the PRP and Cryo-PRP groups compared to Control, with the highest levels observed in the Cryo-PRP group. In contrast, TNF-α expression was significantly reduced following PRP and Cryo-PRP treatment, with minimal staining in the Cryo-PRP group. (D) Quantitative analysis of immunohistochemical staining using integrated optical density per area (IOD/Area × 10 6 ). Data are presented as mean ± SD. * P < 0.05 vs. Control; *** P < 0.001 vs. Control.

    Article Snippet: Subsequently, the sections were incubated overnight at 4 °C with rabbit anti-CD34 antibody (BIOSS, bs-064R; 1:100 dilution).

    Techniques: Immunofluorescence, Immunohistochemical staining, Staining, Control, Expressing